reference/15539544

http://rdf.ncbi.nlm.nih.gov/pubchem/reference/15539544

Outgoing Links

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contentType	Journal Article\|Research Support, Non-U.S. Gov't
endingPage	144
issn	1460-2180 0143-3334
issueIdentifier	1
pageRange	131-144
publicationName	Carcinogenesis
startingPage	131
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bibliographicCitation	Yang L, Zhou J, Meng F, Fu C, Zou X, Liu J, Zhang C, Tan R, Li Z, Guo Q, Wei L. G1 phase cell cycle arrest in NSCLC in response to LZ-106, an analog of enoxacin, is orchestrated through ROS overproduction in a P53-dependent manner. Carcinogenesis. 2019 Mar 12;40(1):131–44. doi: 10.1093/carcin/bgy124. PMID: 30239617.
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date	2018-09-15-04:00^^<http://www.w3.org/2001/XMLSchema#date>
identifier	https://doi.org/10.1093/carcin/bgy124 https://pubmed.ncbi.nlm.nih.gov/30239617
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language	English
source	https://pubmed.ncbi.nlm.nih.gov/ https://www.crossref.org/
title	G1 phase cell cycle arrest in NSCLC in response to LZ-106, an analog of enoxacin, is orchestrated through ROS overproduction in a P53-dependent manner
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Total number of triples: 63.