reference/8055818

http://rdf.ncbi.nlm.nih.gov/pubchem/reference/8055818

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endingPage	604
issn	1756-5901 1756-591X
issueIdentifier	4
pageRange	596-604
publicationName	Metallomics : integrated biometal science
startingPage	596
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bibliographicCitation	Wezynfeld NE, Bonna A, Bal W, Frączyk T. Ni(ii) ions cleave and inactivate human alpha-1 antitrypsin hydrolytically, implicating nickel exposure as a contributing factor in pathologies related to antitrypsin deficiency. Metallomics. 2015 Apr;7(4):596–604. doi: 10.1039/c4mt00316k. PMID: 25579632.
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date	201504
identifier	https://pubmed.ncbi.nlm.nih.gov/25579632 https://doi.org/10.1039/c4mt00316k
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language	English
source	https://www.crossref.org/ https://pubmed.ncbi.nlm.nih.gov/
title	Ni(ii) ions cleave and inactivate human alpha-1 antitrypsin hydrolytically, implicating nickel exposure as a contributing factor in pathologies related to antitrypsin deficiency
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