reference/32337244

http://rdf.ncbi.nlm.nih.gov/pubchem/reference/32337244

Outgoing Links

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endingPage	21561
issn	0021-9258
issueIdentifier	31
pageRange	21544-21561
publicationName	The Journal of biological chemistry
startingPage	21544
bibliographicCitation	Edagawa M, Kawauchi J, Hirata M, Goshima H, Inoue M, Okamoto T, Murakami A, Maehara Y, Kitajima S. Role of Activating Transcription Factor 3 (ATF3) in Endoplasmic Reticulum (ER) Stress-induced Sensitization of p53-deficient Human Colon Cancer Cells to Tumor Necrosis Factor (TNF)-related Apoptosis-inducing Ligand (TRAIL)-mediated Apoptosis through Up-regulation of Death Receptor 5 (DR5) by Zerumbone and Celecoxib. Journal of Biological Chemistry. 2014 Aug;289(31):21544–61. doi: 10.1074/jbc.m114.558890.
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date	2014-08-01-04:00^^<http://www.w3.org/2001/XMLSchema#date>
identifier	https://doi.org/10.1074/jbc.m114.558890 https://pubmed.ncbi.nlm.nih.gov/PMC4118115 https://pubmed.ncbi.nlm.nih.gov/24939851
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language	English
source	https://www.crossref.org/ https://pubmed.ncbi.nlm.nih.gov/
title	Role of Activating Transcription Factor 3 (ATF3) in Endoplasmic Reticulum (ER) Stress-induced Sensitization of p53-deficient Human Colon Cancer Cells to Tumor Necrosis Factor (TNF)-related Apoptosis-inducing Ligand (TRAIL)-mediated Apoptosis through Up-regulation of Death Receptor 5 (DR5) by Zerumbone and Celecoxib
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