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contentType Journal Article|Research Support, Non-U.S. Gov't
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issueIdentifier 21
pageRange 1694-1713
publicationName Circulation
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bibliographicCitation Bertero E, Nickel A, Kohlhaas M, Hohl M, Sequeira V, Brune C, Schwemmlein J, Abeßer M, Schuh K, Kutschka I, Carlein C, Münker K, Atighetchi S, Müller A, Kazakov A, Kappl R, von der Malsburg K, van der Laan M, Schiuma AF, Böhm M, Laufs U, Hoth M, Rehling P, Kuhn M, Dudek J, von der Malsburg A, Prates Roma L, Maack C. Loss of Mitochondrial Ca2+ Uniporter Limits Inotropic Reserve and Provides Trigger and Substrate for Arrhythmias in Barth Syndrome Cardiomyopathy. Circulation. 2021 Nov 23;144(21):1694–713. doi: 10.1161/circulationaha.121.053755. PMID: 34648376.
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date 2021-11-23-04:00^^<http://www.w3.org/2001/XMLSchema#date>
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title Loss of Mitochondrial Ca 2+ Uniporter Limits Inotropic Reserve and Provides Trigger and Substrate for Arrhythmias in Barth Syndrome Cardiomyopathy
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