reference/25620649

http://rdf.ncbi.nlm.nih.gov/pubchem/reference/25620649

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endingPage	17950
issn	0027-8424 1091-6490
issueIdentifier	44
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publicationName	Proceedings of the National Academy of Sciences of the United States of America
startingPage	17945
bibliographicCitation	Sugiyama D, Nishikawa H, Maeda Y, Nishioka M, Tanemura A, Katayama I, Ezoe S, Kanakura Y, Sato E, Fukumori Y, Karbach J, Jäger E, Sakaguchi S. Anti-CCR4 mAb selectively depletes effector-type FoxP3 ⁺ CD4 ⁺ regulatory T cells, evoking antitumor immune responses in humans. Proc. Natl. Acad. Sci. U.S.A. 2013 Oct 14;110(44):17945–50. doi: 10.1073/pnas.1316796110.
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date	2013-10-14-04:00^^<http://www.w3.org/2001/XMLSchema#date>
identifier	https://pubmed.ncbi.nlm.nih.gov/24127572 https://doi.org/10.1073/pnas.1316796110 https://pubmed.ncbi.nlm.nih.gov/PMC3816454
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language	English
source	https://www.crossref.org/ https://pubmed.ncbi.nlm.nih.gov/
title	Anti-CCR4 mAb selectively depletes effector-type FoxP3 + CD4 + regulatory T cells, evoking antitumor immune responses in humans
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