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Object |
contentType |
Journal Article|Research Support, Non-U.S. Gov't|Research Support, U.S. Gov't, P.H.S. |
endingPage |
37335 |
issn |
0021-9258 |
issueIdentifier |
39 |
pageRange |
37330-37335 |
publicationName |
The Journal of biological chemistry |
startingPage |
37330 |
hasFundingAgency |
http://rdf.ncbi.nlm.nih.gov/pubchem/organization/MD5_916de93fe8f49855f42e16db43537393 |
isSupportedBy |
http://rdf.ncbi.nlm.nih.gov/pubchem/grant/MD5_c15ead45edb7bf70fef1bcf5ab9c6cb3 |
bibliographicCitation |
da Costa CA, Masliah E, Checler F. Beta-synuclein displays an antiapoptotic p53-dependent phenotype and protects neurons from 6-hydroxydopamine-induced caspase 3 activation: cross-talk with alpha-synuclein and implication for Parkinson's disease. J Biol Chem. 2003 Sep 26;278(39):37330–5. doi: 10.1074/jbc.m306083200. PMID: 12867415. |
creator |
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date |
2003-09-26-04:00^^<http://www.w3.org/2001/XMLSchema#date> |
identifier |
https://pubmed.ncbi.nlm.nih.gov/12867415 https://doi.org/10.1074/jbc.m306083200 |
isPartOf |
https://portal.issn.org/resource/ISSN/0021-9258 http://rdf.ncbi.nlm.nih.gov/pubchem/journal/4559 |
language |
English |
source |
https://pubmed.ncbi.nlm.nih.gov/ https://www.crossref.org/ |
title |
Beta-synuclein displays an antiapoptotic p53-dependent phenotype and protects neurons from 6-hydroxydopamine-induced caspase 3 activation: cross-talk with alpha-synuclein and implication for Parkinson's disease |
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