reference/17491319

http://rdf.ncbi.nlm.nih.gov/pubchem/reference/17491319

Outgoing Links

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contentType	Journal Article\|Research Support, Non-U.S. Gov't
endingPage	323
issn	0304-3835
issueIdentifier	2
pageRange	313-323
publicationName	Cancer Letters
startingPage	313
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bibliographicCitation	Zhou J, Zhu G, Huang J, Li L, Du Y, Gao Y, Wu D, Wang X, Hsieh JT, He D, Wu K. Non-canonical GLI1/2 activation by PI3K/AKT signaling in renal cell carcinoma: A novel potential therapeutic target. Cancer Lett. 2016 Jan 28;370(2):313–23. doi: 10.1016/j.canlet.2015.11.006. PMID: 26577809.
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date	2016-01-28-04:00^^<http://www.w3.org/2001/XMLSchema#date>
identifier	https://pubmed.ncbi.nlm.nih.gov/26577809 https://doi.org/10.1016/j.canlet.2015.11.006
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language	English
source	https://pubmed.ncbi.nlm.nih.gov/ https://www.crossref.org/
title	Non-canonical GLI1/2 activation by PI3K/AKT signaling in renal cell carcinoma: A novel potential therapeutic target
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