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Journal Article|Review |
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329 |
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1520-5010
0893-228X |
| issueIdentifier |
3 |
| pageRange |
325-329 |
| publicationName |
Chemical Research in Toxicology |
| startingPage |
325 |
| bibliographicCitation |
Pogocki D. Mutation of the Phe20Residue in Alzheimer's Amyloid β-Peptide Might Decrease Its Toxicity Due to Disruption of the Met35−Cupric Site Electron Transfer Pathway. Chem. Res. Toxicol. 2004 Feb 05;17(3):325–9. doi: 10.1021/tx030044w. |
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http://rdf.ncbi.nlm.nih.gov/pubchem/author/MD5_3a1276e5a445f06963f2f37836b6934c |
| date |
2004-02-05-04:00^^<http://www.w3.org/2001/XMLSchema#date> |
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https://doi.org/10.1021/tx030044w
https://pubmed.ncbi.nlm.nih.gov/15025502 |
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https://portal.issn.org/resource/ISSN/1520-5010
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| language |
English |
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https://www.crossref.org/
https://pubmed.ncbi.nlm.nih.gov/ |
| title |
Mutation of the Phe20Residue in Alzheimer's Amyloid β-Peptide Might Decrease Its Toxicity Due to Disruption of the Met35−Cupric Site Electron Transfer Pathway |
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