reference/11819581

http://rdf.ncbi.nlm.nih.gov/pubchem/reference/11819581

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contentType	Journal Article\|Research Support, Non-U.S. Gov't\|Research Support, U.S. Gov't, P.H.S.
endingPage	39
issn	0014-4886
issueIdentifier	1
pageRange	28-39
publicationName	Experimental Neurology
startingPage	28
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bibliographicCitation	Kruman II, Pedersen WA, Springer JE, Mattson MP. ALS-linked Cu/Zn-SOD mutation increases vulnerability of motor neurons to excitotoxicity by a mechanism involving increased oxidative stress and perturbed calcium homeostasis. Exp Neurol. 1999 Nov;160(1):28–39. doi: 10.1006/exnr.1999.7190. PMID: 10630188.
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date	199911
identifier	https://doi.org/10.1006/exnr.1999.7190 https://pubmed.ncbi.nlm.nih.gov/10630188
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language	English
source	https://pubmed.ncbi.nlm.nih.gov/ https://www.crossref.org/
title	ALS-Linked Cu/Zn–SOD Mutation Increases Vulnerability of Motor Neurons to Excitotoxicity by a Mechanism Involving Increased Oxidative Stress and Perturbed Calcium Homeostasis
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