http://rdf.ncbi.nlm.nih.gov/pubchem/patent/WO-03100027-A2

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assignee http://rdf.ncbi.nlm.nih.gov/pubchem/patentassignee/MD5_78587acbfc8d4c9e9ac3baa7021087d7
http://rdf.ncbi.nlm.nih.gov/pubchem/patentassignee/MD5_3b7edd3693b55c0fd41df267c299f173
classificationCPCAdditional http://rdf.ncbi.nlm.nih.gov/pubchem/patentcpc/A61K2039-5154
http://rdf.ncbi.nlm.nih.gov/pubchem/patentcpc/A61K2039-5156
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http://rdf.ncbi.nlm.nih.gov/pubchem/patentcpc/A61K39-00
classificationCPCInventive http://rdf.ncbi.nlm.nih.gov/pubchem/patentcpc/C07K14-78
classificationIPCAdditional http://rdf.ncbi.nlm.nih.gov/pubchem/patentipc/A61K38-00
http://rdf.ncbi.nlm.nih.gov/pubchem/patentipc/A61K39-00
classificationIPCInventive http://rdf.ncbi.nlm.nih.gov/pubchem/patentipc/C07K14-78
filingDate 2003-05-28-04:00^^<http://www.w3.org/2001/XMLSchema#date>
inventor http://rdf.ncbi.nlm.nih.gov/pubchem/patentinventor/MD5_b22427779cc5d516ddaca69c9e2d1af7
publicationDate 2003-12-04-04:00^^<http://www.w3.org/2001/XMLSchema#date>
publicationNumber WO-03100027-A2
titleOfInvention Mutant fibronectin and tumor metastasis
abstract The present invention relates to a mutated fibronectin as a class II-restricted tumor antigen recognized by tumor-reactive CD4+ T cells. In a specific embodiment, the mutation in fibronectin is responsible for the loss of FN matrix formation, leading to the enhanced migration of tumor cells. This provides an exemplary important immune target for effective cancer immunotherapy.
isCitedBy http://rdf.ncbi.nlm.nih.gov/pubchem/patent/DE-102005041616-B4
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priorityDate 2002-05-28-04:00^^<http://www.w3.org/2001/XMLSchema#date>
type http://data.epo.org/linked-data/def/patent/Publication

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