http://rdf.ncbi.nlm.nih.gov/pubchem/patent/US-7536224-B2

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assignee http://rdf.ncbi.nlm.nih.gov/pubchem/patentassignee/MD5_f34119b232d292c72e632b62008eca90
classificationCPCInventive http://rdf.ncbi.nlm.nih.gov/pubchem/patentcpc/A61N1-3622
classificationIPCInventive http://rdf.ncbi.nlm.nih.gov/pubchem/patentipc/A61N1-362
filingDate 2003-04-30-04:00^^<http://www.w3.org/2001/XMLSchema#date>
grantDate 2009-05-19-04:00^^<http://www.w3.org/2001/XMLSchema#date>
inventor http://rdf.ncbi.nlm.nih.gov/pubchem/patentinventor/MD5_34ceb44576b8e0d9dd2815660d0ce7d8
http://rdf.ncbi.nlm.nih.gov/pubchem/patentinventor/MD5_718ebdd52d493cce630df1bcbbe63d3d
http://rdf.ncbi.nlm.nih.gov/pubchem/patentinventor/MD5_d5c2cb9e8988783a1ee9b36d69225d07
publicationDate 2009-05-19-04:00^^<http://www.w3.org/2001/XMLSchema#date>
publicationNumber US-7536224-B2
titleOfInvention Method for elimination of ventricular pro-arrhythmic effect caused by atrial therapy
abstract A system and method are provided for controlling atrial anti-tachycardia pacing (ATP) delivery based on detection of ventricular pro-arrhythmia during or immediately after atrial ATP. Ventricular pro-arrhythmia is detected based on one or more criteria relating to pro-arrhythmic changes including, but not limited to, ventricular rate changes, R-wave morphology changes, and/or 1:1 or nearly 1:1 atrial-ventricular conduction patterns persisting at high ventricular rates. Upon detecting ventricular pro-arrhythmia, a current atrial ATP sequence is aborted. Atrial ATP therapies may subsequently be temporarily or permanently disabled.
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