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filingDate 2021-03-04-04:00^^<http://www.w3.org/2001/XMLSchema#date>
inventor http://rdf.ncbi.nlm.nih.gov/pubchem/patentinventor/MD5_e7539ca4a7dba7bbcb5e309c86a78730
http://rdf.ncbi.nlm.nih.gov/pubchem/patentinventor/MD5_ac3f52312afa5fa36e3a878c27eff08e
http://rdf.ncbi.nlm.nih.gov/pubchem/patentinventor/MD5_ef4e7589eed7c90302215860c5ba351d
http://rdf.ncbi.nlm.nih.gov/pubchem/patentinventor/MD5_0a3ce923d0765c331f4e97ea6d532646
http://rdf.ncbi.nlm.nih.gov/pubchem/patentinventor/MD5_9e1236ea2f0d486023a818104e095b1c
http://rdf.ncbi.nlm.nih.gov/pubchem/patentinventor/MD5_06b80a2f3ee55202e61224d8a112e4d0
publicationDate 2021-07-22-04:00^^<http://www.w3.org/2001/XMLSchema#date>
publicationNumber US-2021220475-A1
titleOfInvention Egfr proteolysis targeting chimeric molecules and associated methods of use
abstract The present invention relates to bifunctional compounds, which find utility to degrade and (inhibit) TBK1. In particular, the present invention is directed to compounds, which contain on one end an E3 ubiquitin ligase binding moiety which binds to an E3 ubiquitin ligase and on the other end a moiety which binds TBK1 such that TBK1 is placed in proximity to the ubiquitin ligase to effect degradation (and inhibition) of TBK1. The present invention exhibits a broad range of pharmacological activities associated with compounds according to the present invention, consistent with the degradation/inhibition of TBK1.
priorityDate 2016-12-23-04:00^^<http://www.w3.org/2001/XMLSchema#date>
type http://data.epo.org/linked-data/def/patent/Publication

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Total number of triples: 7603.