http://rdf.ncbi.nlm.nih.gov/pubchem/patent/US-2011319356-A1

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assignee http://rdf.ncbi.nlm.nih.gov/pubchem/patentassignee/MD5_ef5ee91eb506478b06235dcde255ad7f
classificationCPCInventive http://rdf.ncbi.nlm.nih.gov/pubchem/patentcpc/A61P27-02
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classificationIPCInventive http://rdf.ncbi.nlm.nih.gov/pubchem/patentipc/A61P27-02
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filingDate 2011-06-22-04:00^^<http://www.w3.org/2001/XMLSchema#date>
inventor http://rdf.ncbi.nlm.nih.gov/pubchem/patentinventor/MD5_3e1f5763aed01d9c3fd2a10eb6525c3d
publicationDate 2011-12-29-04:00^^<http://www.w3.org/2001/XMLSchema#date>
publicationNumber US-2011319356-A1
titleOfInvention Methods Of Treating Optic Disorders
abstract The present invention relates to methods for treating optic disorders or for reducing or alleviating the signs, symptoms, or pathological conditions related to such optic disorders. In particular, methods are provided for treating optic disorders, or reducing the symptoms thereof, the methods involving the administration of one or more downstream folate compounds and/or methyl-B12. In one particular embodiment, the method comprises administration of L-methylfolate. In other embodiments, the method involves administering both L-methylfolate and methyl-B12. In still further embodiments, the method further involves reducing dietary intake of folic acid. In certain embodiments, the method further involves identifying a subject organism with a malfunction in one or more of the folate or B4 cycles. In certain embodiments, such a malfunction is one or more of the C677T and A1298C mutations. In still further embodiments, the method further involves identifying a subject organism that is deficient in vitamins B-12 and D3 but which possesses excess homocysteine.
isCitedBy http://rdf.ncbi.nlm.nih.gov/pubchem/patent/WO-2017102715-A1
http://rdf.ncbi.nlm.nih.gov/pubchem/patent/CN-112672745-A
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priorityDate 2010-06-25-04:00^^<http://www.w3.org/2001/XMLSchema#date>
type http://data.epo.org/linked-data/def/patent/Publication

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