http://rdf.ncbi.nlm.nih.gov/pubchem/patent/US-2008058289-A1

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assignee http://rdf.ncbi.nlm.nih.gov/pubchem/patentassignee/MD5_e55f45398cbe9bfb067a51140c0691cd
classificationCPCInventive http://rdf.ncbi.nlm.nih.gov/pubchem/patentcpc/A61K31-66
classificationIPCInventive http://rdf.ncbi.nlm.nih.gov/pubchem/patentipc/A61K31-66
filingDate 2006-08-30-04:00^^<http://www.w3.org/2001/XMLSchema#date>
inventor http://rdf.ncbi.nlm.nih.gov/pubchem/patentinventor/MD5_8c002727de86ba7035476e30185ef1ff
http://rdf.ncbi.nlm.nih.gov/pubchem/patentinventor/MD5_1b84a6afe7deffc8c0d78e4a5f529caa
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publicationDate 2008-03-06-04:00^^<http://www.w3.org/2001/XMLSchema#date>
publicationNumber US-2008058289-A1
titleOfInvention Increasing atp availability by inhibition of creatine kinase (ck) leakage resulting from high-intensity exercise
abstract The present invention relates to a method of increasing Adenosine Triphosphate levels in a mammal wherein 2-(carbamimidoyl-methyl-amino)ethoxyphosphonic acid inhibits the leakage of the Adenosine Triphosphate-dependant enzyme Creatine Kinase. Via the inhibition of Creatine Kinase leakage, Adenosine Triphosphate is not utilized in the conversion of creatine to phosphocreatine, thereby making it available for use in muscular contractions. The increase in availability leads to longer endurance and more forceful muscular contractions.
priorityDate 2006-08-30-04:00^^<http://www.w3.org/2001/XMLSchema#date>
type http://data.epo.org/linked-data/def/patent/Publication

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