http://rdf.ncbi.nlm.nih.gov/pubchem/patent/MX-2008004901-A

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assignee http://rdf.ncbi.nlm.nih.gov/pubchem/patentassignee/MD5_a8cdfbc70d9776e1ea4502b21128b30c
classificationIPCInventive http://rdf.ncbi.nlm.nih.gov/pubchem/patentipc/A61P1-18
http://rdf.ncbi.nlm.nih.gov/pubchem/patentipc/A61P3-10
http://rdf.ncbi.nlm.nih.gov/pubchem/patentipc/A61K39-395
http://rdf.ncbi.nlm.nih.gov/pubchem/patentipc/A61K45-00
filingDate 2006-10-13-04:00^^<http://www.w3.org/2001/XMLSchema#date>
inventor http://rdf.ncbi.nlm.nih.gov/pubchem/patentinventor/MD5_87b69c2a84794f6e9f875c9779be3667
publicationDate 2008-12-16-04:00^^<http://www.w3.org/2001/XMLSchema#date>
publicationNumber MX-2008004901-A
titleOfInvention AGENTS TO DELETE DAMAGE TO TRANSPLANTED ISLOTS AFTER THE ISLOT TRANSPLANT.
abstract An anti-IL-6 receptor antibody is reviewed for its inhibitory effect against transplanted islet dysfunction produced after islet transplantation. As a result, it was discovered that the anti-IL-6 receptor antibody can reduce the transplanted islet dysfunction, improve the survival of the transplanted islet, and also improve the hyperglycemic state of a receptor. It was also discovered that the administration of the anti-IL-6 receptor antibody makes it possible to prevent the production of an inflammatory cytokine in an identification cell after transplantation. Therefore, it was discovered that transplanted islet dysfunction produced after islet transplantation can be prevented using the anti-IL-6 receptor antibody.
priorityDate 2005-10-14-04:00^^<http://www.w3.org/2001/XMLSchema#date>
type http://data.epo.org/linked-data/def/patent/Publication

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http://rdf.ncbi.nlm.nih.gov/pubchem/protein/ACCA0EPY1
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Total number of triples: 584.