http://rdf.ncbi.nlm.nih.gov/pubchem/patent/KR-20130112236-A
Outgoing Links
| Predicate | Object |
|---|---|
| assignee | http://rdf.ncbi.nlm.nih.gov/pubchem/patentassignee/MD5_27c49e2184728df41a493269458e1486 |
| classificationCPCAdditional | http://rdf.ncbi.nlm.nih.gov/pubchem/patentcpc/A23V2200-328 |
| classificationCPCInventive | http://rdf.ncbi.nlm.nih.gov/pubchem/patentcpc/A23L29-035 http://rdf.ncbi.nlm.nih.gov/pubchem/patentcpc/A61K31-22 |
| classificationIPCInventive | http://rdf.ncbi.nlm.nih.gov/pubchem/patentipc/A61P29-00 http://rdf.ncbi.nlm.nih.gov/pubchem/patentipc/A61K31-19 |
| filingDate | 2012-04-03-04:00^^<http://www.w3.org/2001/XMLSchema#date> |
| inventor | http://rdf.ncbi.nlm.nih.gov/pubchem/patentinventor/MD5_8b7656fab4b0b2c066113916127cc92f |
| publicationDate | 2013-10-14-04:00^^<http://www.w3.org/2001/XMLSchema#date> |
| publicationNumber | KR-20130112236-A |
| titleOfInvention | Inhibition of inflammation induced by high glucose containing sodium butyrate as an active ingredient |
| abstract | The present invention relates to a composition for inhibiting inflammation induced by high glucose containing sodium butyrate as an active ingredient. More specifically, sodium butyrate secretes cytokines induced by high glucose such as TNF-α and IL-6. Sodium butyrate by inhibiting NF-kB transcriptional activity, reducing HAT activity, decreasing p300 and acetylated CBP / p30 gene expression, and inducing HDAC activity. It can be seen that it can be usefully used as an active ingredient in the prevention and treatment of inflammatory diseases caused by glucose. |
| isCitedBy | http://rdf.ncbi.nlm.nih.gov/pubchem/patent/CN-112826816-A http://rdf.ncbi.nlm.nih.gov/pubchem/patent/WO-2020058424-A1 |
| priorityDate | 2012-04-03-04:00^^<http://www.w3.org/2001/XMLSchema#date> |
| type | http://data.epo.org/linked-data/def/patent/Publication |
Incoming Links
Total number of triples: 344.