http://rdf.ncbi.nlm.nih.gov/pubchem/patent/KR-20100128761-A

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assignee http://rdf.ncbi.nlm.nih.gov/pubchem/patentassignee/MD5_951ade9cb9f29eb8a29ddae1e2fc87e3
classificationCPCInventive http://rdf.ncbi.nlm.nih.gov/pubchem/patentcpc/A61K9-0019
http://rdf.ncbi.nlm.nih.gov/pubchem/patentcpc/A61K9-20
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classificationIPCInventive http://rdf.ncbi.nlm.nih.gov/pubchem/patentipc/A61P25-28
http://rdf.ncbi.nlm.nih.gov/pubchem/patentipc/A61K35-28
filingDate 2009-05-29-04:00^^<http://www.w3.org/2001/XMLSchema#date>
inventor http://rdf.ncbi.nlm.nih.gov/pubchem/patentinventor/MD5_e4e9ad7ab2a7730cf6b0eb69f12ff247
http://rdf.ncbi.nlm.nih.gov/pubchem/patentinventor/MD5_dd980ad6ce727b73ee9e6ee1b16a96ef
http://rdf.ncbi.nlm.nih.gov/pubchem/patentinventor/MD5_3debfec5c794155d60997d573f2e543e
publicationDate 2010-12-08-04:00^^<http://www.w3.org/2001/XMLSchema#date>
publicationNumber KR-20100128761-A
titleOfInvention New Cell Therapeutic Agent for Improvement of Dementia Symptoms through Immune Regulatory Mechanism of Bone Marrow-derived Mesenchymal Stem Cells
abstract The present invention relates to a composition for treating diseases caused by hyperphosphorylation of tau accumulated in the brain by bone marrow-derived mesenchymal stem cells or toxic effects of active microglia, in particular amnesia or cognitive impairment.n n n The composition of the present invention is made by implanting bone marrow-derived mesenchymal stem cells in the affected area, the bone marrow-derived mesenchymal stem cells of the present composition prevents overphosphorylation of tau and alters the phenotype of microglia to affect the activity of microglia. It is characterized by the treatment of amnesia or cognitive impairment caused by neurodegenerative diseases by preventing the phenomenon causing entanglement of nerve fibers, which are toxic effects.
isCitedBy http://rdf.ncbi.nlm.nih.gov/pubchem/patent/KR-20150026839-A
priorityDate 2009-05-29-04:00^^<http://www.w3.org/2001/XMLSchema#date>
type http://data.epo.org/linked-data/def/patent/Publication

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