http://rdf.ncbi.nlm.nih.gov/pubchem/patent/KR-101289474-B1

Outgoing Links

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classificationCPCAdditional http://rdf.ncbi.nlm.nih.gov/pubchem/patentcpc/A01K2227-105
http://rdf.ncbi.nlm.nih.gov/pubchem/patentcpc/A01K2267-03
http://rdf.ncbi.nlm.nih.gov/pubchem/patentcpc/C12N2015-8527
classificationCPCInventive http://rdf.ncbi.nlm.nih.gov/pubchem/patentcpc/A01K67-0275
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classificationIPCInventive http://rdf.ncbi.nlm.nih.gov/pubchem/patentipc/A01K67-027
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filingDate 2011-03-30-04:00^^<http://www.w3.org/2001/XMLSchema#date>
grantDate 2013-07-25-04:00^^<http://www.w3.org/2001/XMLSchema#date>
publicationDate 2013-07-25-04:00^^<http://www.w3.org/2001/XMLSchema#date>
publicationNumber KR-101289474-B1
titleOfInvention Transgenic mice useful for ephrin-A5 gene function research and methods of making the same
abstract The present invention discloses transgenic mice useful for ephrin-A5 gene function studies and methods of making the same. Specifically, the present invention includes a reporter gene whose expression is regulated by a promoter and enhancer of ephrin-A5 and an ephrin-A5-Fc gene located below the reporter gene, wherein the reporter gene is removed by homologous recombination. By including a recombinant sequence at both ends of the reporter gene so that it can be identified, the region expressed by the expression of the reporter gene Ephrine-A5 gene can be identified, when the reporter gene is removed by homologous recombination Ephrin-A5 Expression of the -Fc gene results in loss or degradation of Ephrine-A5 by binding Ephrine-A5-Fc to the Eph receptor to which Ephrine-A5 binds, resulting in loss or degradation of A5. Disclosed are a transgenic mouse and a method of making the same, which make it possible to identify a phenotype according to the present invention.
priorityDate 2011-03-30-04:00^^<http://www.w3.org/2001/XMLSchema#date>
type http://data.epo.org/linked-data/def/patent/Publication

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