| abstract |
The present invention relates to a method of using a mammal as an attention deficit hyperactivity disorder (ADHD) model, except for a human who has a G protein-coupled receptor kinase interacting protein (GIT1) gene, which is a neuronal synaptic protein in the brain. It is about. In addition, the present invention relates to molecular, cellular, electrobiological, and animal behavioral analysis of GIT1 deficient mice. By monitoring theta waves in the frontal and frontal lobes, we provide a new drug screening method that can lead to the recovery of attention deficit hyperactivity disorder (ADHD). |