http://rdf.ncbi.nlm.nih.gov/pubchem/patent/JP-2019172622-A

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assignee http://rdf.ncbi.nlm.nih.gov/pubchem/patentassignee/MD5_afcfc7af43784847a2171ad49795c3c3
classificationIPCInventive http://rdf.ncbi.nlm.nih.gov/pubchem/patentipc/A61K39-395
http://rdf.ncbi.nlm.nih.gov/pubchem/patentipc/A61K35-35
http://rdf.ncbi.nlm.nih.gov/pubchem/patentipc/A61P1-16
filingDate 2018-03-29-04:00^^<http://www.w3.org/2001/XMLSchema#date>
inventor http://rdf.ncbi.nlm.nih.gov/pubchem/patentinventor/MD5_e1450bc5476bd42274b7decb3c6bc50c
http://rdf.ncbi.nlm.nih.gov/pubchem/patentinventor/MD5_e7e03666da7e222118a46ced9062494d
publicationDate 2019-10-10-04:00^^<http://www.w3.org/2001/XMLSchema#date>
publicationNumber JP-2019172622-A
titleOfInvention Non-alcoholic steatohepatitis therapeutic agent
abstract In non-alcoholic steatohepatitis, exercise and diet therapy and pharmacotherapy are applied as necessary when inflammation is relatively mild. The purpose of this study was to develop a non-alcoholic steatohepatitis therapeutic agent by suppressing tissue fibrosis. The gene expression in a non-alcoholic steatohepatitis model and a simple fatty liver model was compared, and IL17, whose expression was improved in the non-alcoholic steatohepatitis model, was noted. It was confirmed that anti-IL17 antibody suppresses fibrosis of liver tissue in nonalcoholic steatohepatitis, and a nonalcoholic steatohepatitis therapeutic agent containing anti-IL17 antibody was completed. [Selection figure] None
isCitedBy http://rdf.ncbi.nlm.nih.gov/pubchem/patent/CN-112646030-A
priorityDate 2018-03-29-04:00^^<http://www.w3.org/2001/XMLSchema#date>
type http://data.epo.org/linked-data/def/patent/Publication

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