| Predicate |
Object |
| assignee |
http://rdf.ncbi.nlm.nih.gov/pubchem/patentassignee/MD5_44c713790ce00813da2e6c6d57a29456 |
| classificationIPCAdditional |
http://rdf.ncbi.nlm.nih.gov/pubchem/patentipc/C12N15-09 |
| classificationIPCInventive |
http://rdf.ncbi.nlm.nih.gov/pubchem/patentipc/A61K31-7088
http://rdf.ncbi.nlm.nih.gov/pubchem/patentipc/A61K48-00
http://rdf.ncbi.nlm.nih.gov/pubchem/patentipc/A61K45-00
http://rdf.ncbi.nlm.nih.gov/pubchem/patentipc/G01N33-15
http://rdf.ncbi.nlm.nih.gov/pubchem/patentipc/G01N33-50
http://rdf.ncbi.nlm.nih.gov/pubchem/patentipc/A61P35-02
http://rdf.ncbi.nlm.nih.gov/pubchem/patentipc/A61P35-00
http://rdf.ncbi.nlm.nih.gov/pubchem/patentipc/A61P43-00
http://rdf.ncbi.nlm.nih.gov/pubchem/patentipc/C12Q1-37
http://rdf.ncbi.nlm.nih.gov/pubchem/patentipc/C12Q1-02
http://rdf.ncbi.nlm.nih.gov/pubchem/patentipc/A61K38-46 |
| filingDate |
2005-06-21-04:00^^<http://www.w3.org/2001/XMLSchema#date> |
| inventor |
http://rdf.ncbi.nlm.nih.gov/pubchem/patentinventor/MD5_9e86fc64ea777a86f1b0cfaf6b924709
http://rdf.ncbi.nlm.nih.gov/pubchem/patentinventor/MD5_cbcef7fa0038ebe4e664e744d2562c22
http://rdf.ncbi.nlm.nih.gov/pubchem/patentinventor/MD5_ba6c7b458a732e664270e499fc0d4336
http://rdf.ncbi.nlm.nih.gov/pubchem/patentinventor/MD5_41d92619a6c5a3c9ca089fa0d1105549
http://rdf.ncbi.nlm.nih.gov/pubchem/patentinventor/MD5_2f4548e6846f1a063dc1a3e7a3df362d |
| publicationDate |
2006-02-09-04:00^^<http://www.w3.org/2001/XMLSchema#date> |
| publicationNumber |
JP-2006034288-A |
| titleOfInvention |
Method for evaluating substance capable of acting on endoplasmic reticulum stress-induced and / or amyloid-β-induced apoptosis |
| abstract |
The present invention provides a means capable of acting (for example, suppressing or promoting) apoptosis based on a pathway different from the apoptosis signaling pathway in mitochondria. A method for evaluating a substance capable of acting on endoplasmic reticulum stress-induced apoptosis and / or amyloid-beta-induced apoptosis, an evaluation system for the substance, and the endoplasmic reticulum stress-induced apoptosis and / or amyloid-beta-induced apoptosis. The pharmaceutical composition which can inhibit the disease caused in connection with or the progression thereof. [Selection figure] None |
| priorityDate |
2004-06-23-04:00^^<http://www.w3.org/2001/XMLSchema#date> |
| type |
http://data.epo.org/linked-data/def/patent/Publication |