http://rdf.ncbi.nlm.nih.gov/pubchem/patent/EP-2068856-A2

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classificationCPCInventive http://rdf.ncbi.nlm.nih.gov/pubchem/patentcpc/A61K31-54
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classificationIPCInventive http://rdf.ncbi.nlm.nih.gov/pubchem/patentipc/A61K31-17
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filingDate 2007-07-25-04:00^^<http://www.w3.org/2001/XMLSchema#date>
inventor http://rdf.ncbi.nlm.nih.gov/pubchem/patentinventor/MD5_e0cdcd6cee7b93236fa4e9b4a08a363a
http://rdf.ncbi.nlm.nih.gov/pubchem/patentinventor/MD5_78c53420c24b65eec0b8fbddd0e4f778
http://rdf.ncbi.nlm.nih.gov/pubchem/patentinventor/MD5_b19648eff150b87af0202daa8aa09b7e
publicationDate 2009-06-17-04:00^^<http://www.w3.org/2001/XMLSchema#date>
publicationNumber EP-2068856-A2
titleOfInvention Antagonists of endothelial differentiation gene subfamily 3 (edg-3, s1p3) receptors for prevention and treatment of ocular disorders
abstract Antagonists of S1P3 (Edg-3) receptors are provided for attenuation of Smad signaling in a method of down-regulation of receptor signaling and downstream decreased production of connective tissue growth factor in ocular disorders involving CTGF accumulation. Ocular disorders involving inappropriate CTGF accumulation include ocular hypertension, glaucoma, glaucomatous retinopathy, optic neuropathy, macular degeneration, diabetic retinopathy, choroidal neovascularization, proliferative vitreoretinopathy and ocular wound healing, for example. Such disorders are treated by administering antagonists of the present invention.
priorityDate 2006-07-25-04:00^^<http://www.w3.org/2001/XMLSchema#date>
type http://data.epo.org/linked-data/def/patent/Publication

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