http://rdf.ncbi.nlm.nih.gov/pubchem/patent/EP-1913949-A1
Outgoing Links
| Predicate | Object |
|---|---|
| assignee | http://rdf.ncbi.nlm.nih.gov/pubchem/patentassignee/MD5_bbe675b46d686407d64e31a937562663 |
| classificationCPCInventive | http://rdf.ncbi.nlm.nih.gov/pubchem/patentcpc/A61K33-36 http://rdf.ncbi.nlm.nih.gov/pubchem/patentcpc/A61P9-04 |
| classificationIPCInventive | http://rdf.ncbi.nlm.nih.gov/pubchem/patentipc/A61P9-04 http://rdf.ncbi.nlm.nih.gov/pubchem/patentipc/A61K33-36 |
| filingDate | 2006-10-11-04:00^^<http://www.w3.org/2001/XMLSchema#date> |
| inventor | http://rdf.ncbi.nlm.nih.gov/pubchem/patentinventor/MD5_f0e8adf61c2b8d07bffe4644c958ea4d http://rdf.ncbi.nlm.nih.gov/pubchem/patentinventor/MD5_5a9148a99f772e84d99400bfa3a08915 |
| publicationDate | 2008-04-23-04:00^^<http://www.w3.org/2001/XMLSchema#date> |
| publicationNumber | EP-1913949-A1 |
| titleOfInvention | Modulation of substrate preference by selective translocation of the major cardiac glucose transporter Glut4 |
| abstract | This invention is in the field of molecular medicine and provides new leads for anti-diabetic therapies. More in particular it relates to a method for specifically increasing glucose uptake in a muscle cell wherein a stimulus is provided to said muscle cell upon which stimulus the major cardiac glucose transporter GLUT4 is translocated from the GLUT4 storage compartment directly to the sarcolemma, thereby by-passing the insulin-responsive recycling endosomes, wherein said stimulus does not alter the uptake of long-chain fatty acids in said muscle cell. |
| isCitedBy | http://rdf.ncbi.nlm.nih.gov/pubchem/patent/US-2013115633-A1 http://rdf.ncbi.nlm.nih.gov/pubchem/patent/US-9182413-B2 http://rdf.ncbi.nlm.nih.gov/pubchem/patent/WO-2011089392-A1 |
| priorityDate | 2006-10-11-04:00^^<http://www.w3.org/2001/XMLSchema#date> |
| type | http://data.epo.org/linked-data/def/patent/Publication |
Incoming Links
Total number of triples: 1325.