| Predicate |
Object |
| assignee |
http://rdf.ncbi.nlm.nih.gov/pubchem/patentassignee/MD5_10d466a9fac96c33267bd561843b72f3 |
| classificationCPCInventive |
http://rdf.ncbi.nlm.nih.gov/pubchem/patentcpc/A61P3-00
http://rdf.ncbi.nlm.nih.gov/pubchem/patentcpc/A61P3-06
http://rdf.ncbi.nlm.nih.gov/pubchem/patentcpc/A61P3-10
http://rdf.ncbi.nlm.nih.gov/pubchem/patentcpc/A61K31-00
http://rdf.ncbi.nlm.nih.gov/pubchem/patentcpc/A61K31-426 |
| classificationIPCInventive |
http://rdf.ncbi.nlm.nih.gov/pubchem/patentipc/A61K38-55
http://rdf.ncbi.nlm.nih.gov/pubchem/patentipc/A61P3-10
http://rdf.ncbi.nlm.nih.gov/pubchem/patentipc/A61P3-06
http://rdf.ncbi.nlm.nih.gov/pubchem/patentipc/A61K31-426
http://rdf.ncbi.nlm.nih.gov/pubchem/patentipc/A61K31-425
http://rdf.ncbi.nlm.nih.gov/pubchem/patentipc/A61K31-40
http://rdf.ncbi.nlm.nih.gov/pubchem/patentipc/A61K31-00 |
| filingDate |
2002-08-09-04:00^^<http://www.w3.org/2001/XMLSchema#date> |
| inventor |
http://rdf.ncbi.nlm.nih.gov/pubchem/patentinventor/MD5_4f5e4daa0a864c08047e0178abe599e8
http://rdf.ncbi.nlm.nih.gov/pubchem/patentinventor/MD5_e27dab0574ee22beeda7d1e3b63a4f38
http://rdf.ncbi.nlm.nih.gov/pubchem/patentinventor/MD5_36d1544cc8153cc6f947c6dc57c45926
http://rdf.ncbi.nlm.nih.gov/pubchem/patentinventor/MD5_aef4c6fa943e0879ae710ff730655cd3 |
| publicationDate |
2005-05-11-04:00^^<http://www.w3.org/2001/XMLSchema#date> |
| publicationNumber |
EP-1528931-A1 |
| titleOfInvention |
Methods for improving islet signaling in diabetes mellitus and for its prevention |
| abstract |
The present invention discloses methods for therapeutically treating mammals, including but not limited to humans, to increase the relative insulin producing performance of endogenous pancreatic beta -cells, to cause differentiation of pancreatic epithelial cells into insulin producing beta -cells, to improve muscle sensitivity to insulin and other weight control efforts by the chronic oral administration ofa DP N-inhibitor. The administration causes the active form of GLP-l and other non-nutrient stimulated growth hormones to remain biologically active longer under physiological conditions. The extended presence of such hormones, in particular in the pancreatic tissue can also facilitate differentiation and regeneration of the J3-cells already present that are in need of repair. |
| priorityDate |
2002-08-09-04:00^^<http://www.w3.org/2001/XMLSchema#date> |
| type |
http://data.epo.org/linked-data/def/patent/Publication |