http://rdf.ncbi.nlm.nih.gov/pubchem/patent/CN-103751174-B
Outgoing Links
| Predicate | Object |
|---|---|
| classificationIPCInventive | http://rdf.ncbi.nlm.nih.gov/pubchem/patentipc/A61P1-16 http://rdf.ncbi.nlm.nih.gov/pubchem/patentipc/A61K31-357 http://rdf.ncbi.nlm.nih.gov/pubchem/patentipc/A61K31-36 http://rdf.ncbi.nlm.nih.gov/pubchem/patentipc/A61K31-09 |
| filingDate | 2013-12-12-04:00^^<http://www.w3.org/2001/XMLSchema#date> |
| grantDate | 2016-03-02-04:00^^<http://www.w3.org/2001/XMLSchema#date> |
| publicationDate | 2016-03-02-04:00^^<http://www.w3.org/2001/XMLSchema#date> |
| publicationNumber | CN-103751174-B |
| titleOfInvention | Fructus Schisandrae Chinensis monomeric compound is preparing the application in hepatotoxic medication caused by prevention and therapy acetaminophen |
| abstract | The open Fructus Schisandrae Chinensis monomeric compound of the present invention is preparing the application in hepatotoxic medication caused by prevention and therapy APAP, and described Fructus Schisandrae Chinensis monomeric compound is deoxyschizandrin, schisandrin C, schisandrin or schisantherin A.Found through experiments alanine aminotransferase (ALT) and aspartate aminotransferase (AST) content that the relevant monomeric compound of described Fructus Schisandrae Chinensis significantly can reduce liver injury model animal caused by APAP; suppress the consumption of reductive glutathione in hepatocyte; and effectively improve hepatic necrosis degree, experimental result shows that Fructus Schisandrae Chinensis monomeric compound has significant treatment and protected effect to the liver toxicity symptom that APAP causes. |
| priorityDate | 2013-12-12-04:00^^<http://www.w3.org/2001/XMLSchema#date> |
| type | http://data.epo.org/linked-data/def/patent/Publication |
Incoming Links
Total number of triples: 451.