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assignee http://rdf.ncbi.nlm.nih.gov/pubchem/patentassignee/MD5_ce384cea17765179fd01cd5c80f294ef
classificationCPCAdditional http://rdf.ncbi.nlm.nih.gov/pubchem/patentcpc/G01N2800-122
classificationCPCInventive http://rdf.ncbi.nlm.nih.gov/pubchem/patentcpc/G01N33-5044
classificationIPCInventive http://rdf.ncbi.nlm.nih.gov/pubchem/patentipc/C12Q1-02
filingDate 2005-05-24-04:00^^<http://www.w3.org/2001/XMLSchema#date>
inventor http://rdf.ncbi.nlm.nih.gov/pubchem/patentinventor/MD5_325b1087fc770efc20afbdbe26912846
http://rdf.ncbi.nlm.nih.gov/pubchem/patentinventor/MD5_4d8aa19f51b45469be51e7d656b9d3e2
http://rdf.ncbi.nlm.nih.gov/pubchem/patentinventor/MD5_443ecb503e7843e22644311ed4208a3d
publicationDate 2006-11-24-04:00^^<http://www.w3.org/2001/XMLSchema#date>
publicationNumber CA-2508256-A1
titleOfInvention Screening assay for improvement of epithelial barrier function
abstract The present invention relates to the use of an air-liquid interface culture model using asthmatic bronchial epithelial cells, which exhibit impaired epithelial barrier function. It is proposed to use asthmatic epithelial cultures, in the absence of added Th2 or proinflammatory cytokines such as IL-13, as an in vitro model to screen for agents that can act to improve impaired asthmatic epithelial barrier function. Such agents may have therapeutic utility in asthma patients.
isCitedBy http://rdf.ncbi.nlm.nih.gov/pubchem/patent/DE-202016000904-U1
priorityDate 2005-05-24-04:00^^<http://www.w3.org/2001/XMLSchema#date>
type http://data.epo.org/linked-data/def/patent/Publication

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